Pam Reynolds' NDE--the "clicks": Continuous or discontinuous?

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Let's try to stick to the maxim "Play the ball, not the man", guys, hey? "Your post was unresponsive and you did not seem to recognise my points. There is no point in responding" is fine. (I would be a hypocrite if I wrote otherwise because I have written similar responses myself). "Your IQ is barely above average" and "You are a moron" stretch our #1 rule of respect and no personal attacks past its breaking point.
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(2019-07-24, 12:44 AM)tim Wrote: Moderators: I can't seem to make a post with normal sized format.

I've edited your posts to fix them, tim. FYI, I did this by clicking the "View source" button in the editor (it's the icon at the far right of the bottom of the toolbar, a piece of paper with a folded over right corner), and then deleting the opening and closing "size", "font", and "color" tags (enclosed in square brackets - the closing ones with a preceding forward slash).
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(2019-07-24, 06:15 AM)ParapsychResearcher Wrote: Also, if it's an option, please lock this thread.

Done.
(2019-07-24, 06:57 AM)Laird Wrote: Done.

And reverted per discussion in the thread Max_B started: https://psiencequest.net/forums/thread-locking-threads
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(2019-07-22, 01:14 PM)tim Wrote: ...
Burst suppression flattens brainwaves. That’s simply a fact. The ratio of burst to suppression, varies according to the dosage of barbiturates (then). See here in the paragraph below.

Therapy (barbiturate, propofol, or halogenated anesthetic) is titrated to an electroencephalographic (cEEG) endpoint. Complete pharmacologic suppression results in a flat-line EEG. Typically, a 1:10 burst to suppression ratio is chosen as an arbitrary endpoint, but this is neither evidence based nor a universal practice. In other words, a 10 second screen of EEG would have 1 second of burst activity and 9 seconds of flat-line EEG. Optimal dosing is unknown and there is no evidence base to guide therapy (6).

https://www.openanesthesia.org/burst_suppression/

...

As this thread has been reopened, I'll ask one question if I may.

Is it known what the burst suppression ratio was in the case of Pam Reynolds?

To avoid any misunderstanding about my reason for asking this, it's because there is some experimental literature on the relationship between the burst suppression ratio and the depth of anaesthesia. If it's known what the BSR was in this case, it may be possible to reach some kind of evidence-based conclusion about what the effect of burst suppression would have been. I hope people agree that would be in everyone's interests.
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(2019-07-25, 07:44 AM)Chris Wrote: As this thread has been reopened, I'll ask one question if I may.

Is it known what the burst suppression ratio was in the case of Pam Reynolds?

To avoid any misunderstanding about my reason for asking this, it's because there is some experimental literature on the relationship between the burst suppression ratio and the depth of anaesthesia. If it's known what the BSR was in this case, it may be possible to reach some kind of evidence-based conclusion about what the effect of burst suppression would have been. I hope people agree that would be in everyone's interests.
Would be awesome, we should ask somebody who had access to medical records.
(2019-07-25, 08:18 AM)Raf999 Wrote: Would be awesome, we should ask somebody who had access to medical records.

For all I know, it may already be in the published literature about the case.
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(2019-07-25, 08:26 AM)Chris Wrote: For all I know, it may already be in the published literature about the case.

It is - see our book The Self Does Not Die, in particular chapter 11 with an addendum by Dr Karl Greene, the neurosurgeon who assisted Dr Robert Spetzler, the lead surgeon who operated Pam Reynolds. Greene has responded to several relevant questions.

Smithy
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